human pancreatic cancer cell lines aspc 1 (ATCC)
ATCC is a verified supplier 
ATCC manufactures this product 
99
Structured Review
ATCC
human pancreatic cancer cell lines aspc 1
Human Pancreatic Cancer Cell Lines Aspc 1, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 3276 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+pancreatic+cancer+cell+lines+aspc+1/AsPC-1/pm42054368-216-0-19
Average 99 stars, based on 3276 article reviews
Human Pancreatic Cancer Cell Lines Aspc 1, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 3276 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/human+pancreatic+cancer+cell+lines+aspc+1/AsPC-1/pm42054368-216-0-19
Average 99 stars, based on 3276 article reviews
human pancreatic cancer cell lines aspc 1 - by Bioz Stars,
2026-09
99/100 stars
Images
Related Articles
Cell Culture:Article Title: Fra-2 controls the response to the KRAS inhibitor MRTX-1133 in pancreatic ductal adenocarcinoma. Article Snippet: KRAS mutations are present in the majority of pancreatic ductal adenocarcinomas (PDAC) and are key drivers of tumor progression.. Although mutation-specific KRAS inhibitors (KRASi) have shown encouraging clinical activity, their therapeutic benefit is limited by the insurgence of resistance.. Here, we hypothesized that the stress-response mediator Fra-2 contributes to this resistance. Article Title: SQLE-catalyzed squalene metabolism promotes mitochondrial biogenesis and tumor development in K-ras-driven cancer. Article Snippet: It is well known that activation of oncogenic K-ras alone is insufficient to drive tumor development and that additional factors are needed for full malignant transformation, but the metabolic pathways and regulatory mechanisms that facilitate K-ras-driven cancer development remain to be characterized.. Here we show that SQLE, a key enzyme in cholesterol synthesis, is upregulated in K-ras-driven cancer and its high expression is correlated with poor clinical outcome.. K-ras regulates SQLE expression in a biphasic manner through reactive oxygen species and MYC signaling. |